Why is vitamin B deficiency common? What are the symptoms?
Vitamin B deficiency is common, primarily because it is a water-soluble vitamin that cannot be stored in the body⁽¹⁾. This means we need to consume enough vitamin B daily through our diet, and inadequate dietary intake or certain lifestyle factors may lead to deficiencies⁽²⁾.
Additionally, certain groups are more prone to vitamin B deficiency, including:
Vegetarians: Since vitamin B is primarily found in animal products⁽³⁾.
Excessive alcohol consumers: Alcohol interferes with the absorption and utilization of vitamin B.
Pregnant and breastfeeding women: Due to increased nutritional demands.
Older adults: Absorption efficiency decreases with age.
Processed and genetically modified foods: Nutritional content may be altered or reduced.
Have you noticed any signs of vitamin B deficiency in yourself? Below are the primary functions of vitamin B1 (thiamine) and the potential effects of its deficiency:
Thiamine involve in below functions, Thiamine deficiency affects
the energy metabolism :
Citric Acid Cycle - generating energy rich molecules ATP
Carbohydrate metabolism - helps convert glucose for energy production
the neuronal-transmitter synthesis:
synthesis of Acetylchroline
nervous system function:
Myelin Sheath maintenance and formation
Neurotransmitter Function - regulating brain activity & maintain healthy health system
antioxidant properties:
protect cells from oxidative stress and counteract oxidative damage
cellular growth and development:
involve in DNA replication and cellular division
immune system support:
the health of immune cells and avoid infection
cardiovascular health:
energy supply to the heart muscle
digestive health:
contributing HCL production in stomach
A long-term deficiency of vitamin B1 (thiamine) is one of the contributing factors to Parkinson's disease and various brain disorders. Oxidative stress can increase oxidative damage to brain cells, leading to impaired energy production and the accumulation of toxic byproducts. Oxidative stress damages cellular components, triggers neuroinflammation, and further results in neurological dysfunction and neuronal death.
